ARS2 Antibody from FORTIS LIFE SCIENCES

Search, find, compare suppliers for anti-ARS2 antibody, protein, ELISA kits.

Edit 
Antigenic SpecificityARS2
Clonepolyclonal
Host SpeciesRabbit
Reactive Specieshuman, mouse (predicted: zebrafish, X. laevis, bovine, orangutan)
Isotypen/a
Formataffinity purified
Size10 µl
Concentration1000 µg/ml
ApplicationsWB, IP
Reviews / RatingsIf you have used this antibody, please help fellow researchers by submitting reviews to pAbmAbs and antYbuddY.
DescriptionRabbit anti-ARS2 antibody, affinity purified. Arsenate resistance protein 2 (ARS2) acts as a mediator between the cap-binding complex (CBC) and the primary microRNAs (miRNAs) processing machinery during cell proliferation. ARS2 contributes to the stability and delivery of capped primary miRNA transcripts to the primary miRNA processing complex containing DGCR8 and DROSHA, thereby playing a role in RNA-mediated gene silencing (RNAi) by miRNAs. ARS2 does not directly confer arsenite resistance but rather modulates arsenic sensitivity. Independently of its activity on miRNAs, it is necessary and sufficient to promote neural stem cell self-renewa and does so by directly binding SOX2 promoter and positively regulating its transcription (By similarity) [taken from the Universal Protein Resource (UniProt) www.uniprot.org/uniprot/Q9BXP5].
ImmunogenBetween 1 and 50
Other Namesserrate; serrate RNA effector molecule homolog; ASR2; arsenite-resistance protein 2; arsenate resistance protein ARS2; ARS2; arsenate resistance protein 2; arsenite resistance protein
Gene, Accession #Accession: NP_056992.4, Gene ID: 51593, UniProt: Q9BXP5
Catalog #A304-550A-T
Price$121
Order / More InfoARS2 Antibody from FORTIS LIFE SCIENCES
Product Specific Referencesn/a
FORTIS LIFE SCIENCES
FORTIS LIFE SCIENCES
FORTIS LIFE SCIENCES
1440 Main Street, Suite 300
Waltham MA 02451
P: 800.338.9579

info@fortislife.com

https://www.fortislife.com

Profile of FORTIS LIFE SCIENCES
Return to Antibodies

© 1980 - 2024 Linscott's Directory, Linscott's USA. All rights reserved.