SOS1 Antibody from MYBIOSOURCE INC.

Search, find, compare suppliers for anti-SOS1 antibody, protein, ELISA kits.

Edit 
Antigenic SpecificitySOS1
Clone[SOS-01]
Host SpeciesMouse
Reactive Speciesmouse
IsotypeIgG1
Formatunconjugated
Size0.1 mg
ConcentrationIgG concentration 1.0mg/ml
ApplicationsELISA (EIA), Immunofluorescence (IF), Western Blot (WB)
Reviews / RatingsIf you have used this antibody, please help fellow researchers by submitting reviews to pAbmAbs and antYbuddY.
DescriptionMouse anti Human SOS1 antibody, clone SOS-1 recognizes SOS1 (son of sevenless homolog 1), a ubiquitously expressed 1333 amino acid ~150kDa intracellular protein and bifunctional guanine nucleotide exchange factor (GEF), which plays a role in growth and differentiation signalling pathways and in actin reorganization, by acting as a regulator of Ras and Rac/Rho GTPases. Mutations in the SOS1 gene are responsible for the rare oral overgrowth condition known as gingival fibromatosis 1 (Hart et al. 2002), and also the cause of the relatively common autosomal dominant disorder Noonan syndrome type 4 (NS4), characterized by short stature, congenital heart malformation, bleeding diathesis and distinctive facial features (Hanna et al. 2009).
Immunogenn/a
Other Names[son of sevenless homolog 1; Son of sevenless homolog 1; son of sevenless homolog 1; SOS-1; gingival fibromatosis, hereditary, 1; guanine nucleotide exchange factor; son of sevenless homolog 1 (Drosophila)], [SOS1; SOS1; GF1; HGF; NS4; GGF1; GINGF; SOS-1]
Gene, Accession #[SOS1], Gene ID: 6654, NCBI: NP_005624.2, UniProt: Q07889
Catalog #MBS217406
Price$300
Order / More InfoSOS1 Antibody from MYBIOSOURCE INC.
Product Specific Referencesn/a
MYBIOSOURCE INC.
MYBIOSOURCE INC.
MYBIOSOURCE INC.
P.O. Box 153308
San Diego CA 92195-3308
P: 1.858.633.0165
P: 1.888.MBS.0165 (1.888.627.0165) (US & Canada)
F: 1.858.633.0166

sales@mybiosource.com

http://www.MyBioSource.com

Profile of MYBIOSOURCE INC.
Return to Antibodies

© 1980 - 2024 Linscott's Directory, Linscott's USA. All rights reserved.